Obesity and Careful Physical Examination

Child obesity is alarmingly prevalent in the United States today; approximately 18 percent of adolescents are considered obese. Obesity is defined as “one of the most pervasive chronic diseases...that affects more than one-third of the population” (Source 2). While obesity may be a major challenge facing adults in the United States, adolescents are facing a similar struggle. More than 33 percents of all children and teens suffer from obesity. But why is child obesity such a major problem in the United States? Though obesity has been shown to have some genetic inheritance between parents and offspring. Adolescents are regularly eating fatty and unhealthy foods, and are not getting the

Childhood obesity has increased dramatically over the last three decades, and conditions in many communities continue to act as barriers to healthy eating and adequate physical activity. Childhood obesity is a serious health problem that has adverse and potentially long-lasting consequences for individuals, families, and communities. Perhaps most shocking, life expectancy for today’s children may be shortened in the United States because of the impact of childhood obesity (Olshansky and Ludwig, 2005).

Since 1980 the rates of child obesity have more than tripled which has caused a growing pandemic of childhood obesity in the United States. Out of all the young children and adolescents within the age group of two through nineteen about 12.7 million are obese. That is the equivalent of about 17% of America’s population that is suffering from childhood obesity. Childhood obesity is too prevalent in all American households. Childhood obesity is detrimental on a national scale, since it has been growing at a steady rate in the United States of children not reaching the daily-recommended physical activity, the absence of a balanced diet with overconsumption of eating, and more critically the increase of type 2 diabetes.

indicate that inherited genetic variation is an important risk factor for obesity. It was also pointed

After breaking down the components of the biopsychosocial model in respect to childhood obesity, the literature contained some strengths and weaknesses. In the past, heredity and environmental factors were known to operate separately, however research indicates that genes affect psychological and social factors, promoting the biopsychosocial model. The Human Obesity Gene Map, published in 1994 provided research regarding the origin of obesity to mutations in genes, disorders, obesity phenotypes, or heredity. This collection of specific genes is annually updated and provides current information regarding obesity. This is a strength to the ongoing research of the etiology of obesity and how it may cause other

Due to the rapid increase in childhood weight gain, the threat of obesity to our health, environment, and economy is enormous. According to the Economic Cost of Obesity, “The estimated annual health care costs of obesity – related illness are a staggering $190.2 billion or nearly twenty-one percent of annual medical spending in the United States. Childhood obesity alone is responsible for fourteen billion in direct medical costs." (Economic Costs of Obesity 4) Obese kids tend to become obese adults. According to Alliance for a Healthier generation, one in three children (2-19 years) in the United States is overweight or obese. There is evidence that a child’s environment has a great impact on childhood obesity. Children are being raised

There is also continued increase in the proportion of children at risk of being overweight (Puhl & Latner, 2007). Childhood and adolescent obesity and overweight trend increased considerably between 1999 and 2004. However, the trend seemingly leveled between 2005 and 2006, and then surged in subsequent years. In 2008, the estimated obesity prevalence rate among children and adolescents of 2-19 years of age was 16.3 percent and overweight prevalence rate was 31.9 percent (Fleming et al., 2008). In 2010, it was estimated that 38 percent of children in the European Union and 50 percent of children in North America were overweight. This dramatic increase in childhood obesity is likely to have considerable long-term impact for economics and public health. If not reversed, the public health obesity toll is likely to continue rising as children and adolescents enter adulthood and start experiencing delayed and usually life threatening obesity complications (Fleming et al., 2008). In addition, there is increasing concerns regarding the vulnerability of many children to the adverse emotional and social obesity consequences. The effects of some of these consequences may be immediate with undesirable health outcomes and potentially lasting effects (Puhl & Latner, 2007). Childhood obesity is specifically problematic because it is not only linked to various comorbid physical and psychological problems but also adult obesity predictors and mortality risk factor (Werthmann et al.

According to the Center for Disease Control and Prevention, “Childhood obesity has more than doubled in children and tripled in adolescents in the past 30 years,” (“Childhood Obesity Facts”). The monumental question that researchers seem to be asking is why the increase now? Childhood obesity has become a paramount problem in the United States in recent years due to various social, biological and technological factors that ultimately requires immediate assistance in order to promote a healthier lifestyle for children as they transition into adulthood.

The cause of childhood obesity is widely debated. There are some who believe childhood obesity is caused by socioeconomic factors while others believe it is caused by media marketing junk food to children. As a response to this ongoing debate, food markers, mainly fast food, try to defend themselves by blaming childhood obesity on physical inactivity and the lack of parental influence on a healthy diet. Despite what many researchers may believe, childhood obesity is caused by a combination of key factors. According to a well-written article by Erica Roth, Childhood Obesity, she along with other doctors believes that obesity could be caused by many different factors. Some may include “family history, many psychological factors, and lifestyle” (Childhood Obesity). However, the causes of childhood obesity are not limited to these life threatening factors, childhood obesity is almost always

The hormone Leptin containing a malfunction is one of the causes for Monogenic Obesity, among many others. Leptin is a hormone that helps regulate body fat and also helps in parts of the brain that controls hunger and tells the body when it’s full. When the Leptin hormone is shortened or absent the body doesn’t secrete fat, but rather stores it making a person become obese. In 1997, two Pakistani cousins were reported with extreme obesity, “Both children had undetectable levels of serum leptin and were found to be homozygous for a frameshift mutation in the LEP gene (ΔG133), which resulted in a truncated protein that was not secreted.” (endocrine.org) In other words, the children contained two types of the same gene whether it be dominant or recessive that were either inserted or deleted causing the levels of the Leptin hormone to be shortened. After the findings of the two Pakistani cousins, there have been five more cases reported with children that lack the leptin hormone, which are all of Pakistani descent, but not related to each other. There has only been one case reported that was found in an Algerian family of three siblings in which the leptin receptor was found abnormal were one was joined to the other allowing a mutated one to work causing there to be more Leptin hormones than there needed to

Childhood obesity is one of the major public health challenges of the 21st century. The prevalence of obesity is increasing globally. In 2013, the number of overweight children under the age of five was estimated over 42 million. Childhood obesity can cause premature death and disability in adulthood. Overweight and obese children will grow up to become obese adults and are more likely to develop diseases like cardiovascular diseases and diabetes at a younger age. Many factors can contribute overweight and obesity in children, however a global shift in dietary habits and lack of physical activity play a crucial role. Overweight and obesity are preventable. Unlike adults, children cannot select the environment they live or the food they eat, they are unware about the long term health consequences of their behavior. Therefore, it is important to have strict policies for the prevention of obesity epidemic. School play an important role in fighting against the epidemic of childhood obesity (World Health Organization, 2016). Even after the legislature has enacted laws to support school nutrition and physical education, many states including, Texas has not yet adopted these policies. It is important to have these policies in practice to prevent childhood obesity (National Conference of State Legislatures, 2014). Government play an important role in making sustainable changes in public health. For that reason, the author is intended to

A person is more likely to develop obesity if one or both parents are obese. Genetics also affect hormones involved in fat regulation. For example, one genetic cause of obesity is leptin deficiency. Leptin is a hormone produced in fat cells and also in the placenta. Leptin controls weight by signaling the brain to eat less when body fat stores are too high. If, for some reason, the body cannot produce enough leptin or leptin cannot signal the brain to eat less, this control is lost, and obesity occurs. The role of leptin replacement as a treatment for obesity is currently being

Many people struggling with morbid obesity have been fighting their weight since they were kids. Many people assume that obesity in children is the result of parents not knowing taking control of their child’s eating habits, but this is seldom the sole cause. Many research studies have shown that children typically will grow up to have a similar BMI to their parents, and siblings will have similar BMI’s to one another. Researchers use this data to show that there is a heavy genetic influence on a person’s weight and simply eating too much or not exercising enough is not the sole determinant of someone’s BMI.

Childhood obesity may not seem like a serious problem, but it is a serious medical condition that can have major effects on a child. Although genetics play a role in determining a child’s weight, it is usually due to a child’s amount of exercise and the consumption of healthy meals. Doctors are concerned with the issue, as we all should be, and they are creating new programs that are geared toward helping children learn how to follow a healthier lifestyle. There are some promising outlooks with these programs, and most doctors agree that parents should help their children create a more active and healthy lifestyle. They also agree that the government should provide more attention to the issue as well. Since the growing problem of childhood

Everybody knows the child that can eat any type of food all day and never seem to gain weight. This occurrence has led researchers to investigate the role that genetics plays in childhood obesity. Not all children who are inactive or who eat poorly are obese, much in the same way that some obese children eat fairly healthy, and exercise moderately. Heredity has recently been shown to influence body fat percentage, regional fat storage, and the body?s response to overeating (Rush, 1). Children who have obese parents are 80% more likely to be obese than their lean parented counterparts (Buffington, 16). This familial correlation is contributed to genetics as well as the parents eating habits. Children with obese parents typically aren?t taught the correct way to choose when and what food to eat, leading to poor eating habits and eventually obesity. Many genetic defects can have a significant effect on obesity such as variable thyroid activity and pituitary defects. Abnormalities in any one of these regulators could be responsible for appetite abnormalities and weight gain. Furthermore, obesity leads to defects in appetite regulation, hormone production, and metabolic events (Oklahoma Cooperative, 4) that are responsible for further weight gain,