A neuron releases an excitatory neurotransmitter. Some neurons are stimulated and some are not. How is it possible that the same signal activates one neuron and not the other?
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A neuron releases an excitatory neurotransmitter. Some neurons are stimulated and some are not. How is it possible that the same signal activates one neuron and not the other?
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- Cocaine blocks the removal of dopamine from the synapse, leading to a rapid rise of dopamine in the synapse. Which of the following best describes what will likely occur as a result of increased dopamine in the synapse? The presynaptic cell will be unable to release more neurotransmitter. the postsynaptic cell will return to its resting potential, causing feelings of well-being. The postsynaptic cell will be constantly activated, causing feelings of euphoria. The postsynaptic cell will respond more quickly to a change in output of the presynaptic cell, causing feelings of euphoria. The buildup of neurotransmitter will activate the presynaptic cell, causing feelings of euphoria.Which of the following would occur if a neuron was experimentally stimulated simultaneously at both ends? The action potentials would pass in the middle and travel to the opposite ends The action potentials would stop as they meet in the middle. The stronger action potential would override the weaker action potential. Summation would occur when the action potentials meet in the middle, resulting in a larger action potential.Which of the following are the likely explanations for the failure of a postsynaptic neuron to trigger an action potential? Select all that apply. The EPSPs are neutralized by the neurotransmitter The summation of IPSPs and EPSPs is insufficient to depolarize the cell to threshold The synapses generating the EPSPs are too close to the axon hillock The synapses generating the EPSPs are too far from the axon hillock
- Imagine an experiment in which the neurons pictured here are bathed in a solution containing a nerve poison. The presynaptic neuron is stimulated and produces an action potential, but this does not result in a PSP in the postsynaptic neuron. When the experimenter adds some neurotransmitter to the synapse, the postsynaptic neuron still produces no PSP. How does the poison act to disrupt nerve function?During excitatory synaptic transmission, the electrical signal carried along the axon of a presynaptic neuron causes the release of neurotransmitters into the synapse and its sub- sequent reception by receptors on the postsynaptic neurons. The activation of receptors on the postsynaptic neurons then triggers the generation of the new action potential in that neuron. Both chemical and electrical neuronal signals are mediated by the movement of ions across the neuronal membrane. Which of the following correctly describes the movement of ions across neuronal membranes as an action potential is conducted down the neuron? A B с D At rest, the internal side of the membrane has an overall positive charge, but as the ac- tion potential moves down the neuron, more ions flow out of the neuron to neutralize the membrane. At rest, the internal side of the membrane has an overall positive charge, but as the ac- tion potential moves down the neuron, more ions flow into the neuron making the membrane…For the nervous system to convey information using electrical signals, the signals typically must be passed from one neuron to another. Oftentimes, this transfer of information across the synapse occurs via the release of neurotransmitters from the pre-synaptic terminal. When a large stimulus is detected in one neuron, it often conveys this information by releasing a larger amount of neurotransmitter into the synapse.
- A neurotransmitter can have one of two effects on the postsynaptic neuron. If a neurotransmitter causes an increase in the likelihood that a neuron will send signals than the neurotransmitter is , but if it decreases the likelihood that it will send a signal than the neurotransmitter is : excitatroy : facilitating : impeding : inhibitoryNeuron A is an inhibitory neuron that is activated when the person feels heat in the tip of their index finger. Neuron A sends signals to neuron B (the postsynaptic neuron). First consider how a stimulus impacts the firing rate of neuron A. For example, when the person touches ice with their index finger vs. when the person touches a hot cup of coffee with their index finger? How would each of these impact the frequency of action potentials, the size of action potentials and the amount of neurotransmitter released. Second, consider Drug-D that causes all sodium channels in neurons to close immediately. How would taking Drug-D affect the firing rate of neuron A when the person touches a hot pan? In turn, how would this affect the firing rate of post-synaptic neuron B? Rubric: Impact of heat on A Impact of ice on A ( Impact of drug X on A Impact of drug X on B 1Parkinson's Disease Parkinson's disease is neurodegenerative disorder that affects movement. Most people affected with Parkinson's disease demonstrate rigidity, slow movement, and shaking. The symptoms of Parkinson's disease occur when the cells that produce dopamine neurotransmitters die in the brain. Explain how the signal transmission at a synapse in an individual with Parkinson's disease is different than an unaffected individual. Describe the normal process of signal transmission at a synapse. Start with the arrival of an action potential at the axon terminal and include the name of the neurotransmitter that is affected by Parkinson's disease. Explain how the process is different in individuals affected with Parkinson's disease.
- Which of the following statements regarding postsynaptic potentials is correct? IPSPs are the result of influx of potassium ions and EPSPs are the result of efflux of sodium ions. IPSPs are the result of efflux of potassium ions and EPSPs are the result of influx of chloride ions. IPSPs are the result of efflux of potassium ions and EPSPs are the result of influx of sodium ions. IPSPs are the result of efflux of chloride ions and EPSPs are the result of efflux of calcium ions.In the laboratory, researchers can apply an electrical stimulus at any point along the axon, making action potentials travel in both directions from the point of stimulation. An action potential moving in the usual direction, away from the axon hillock, is said to be traveling in the orthodromic direction. An action potential traveling toward the axon hillock is traveling in the antidromic direction. If we started an orthodromic action potential at the axon hillock and an antidromic action potential at the opposite end of the axon, what would happen when they met at the center? Why?Which of the following is NOT true regarding the changes that occur during an action potential. Check All That Apply When stimulus is sufficient to cause the threshold potential to be reached, the area stimulated opens its voltage-gated sodium channels.When stimulus is sufficient to cause the threshold potential to be reached, the area stimulated opens its voltage-gated sodium channels. After threshold is reached, the sodium ions move into the cell via the Na+/K+ pump, the inside of the cell becomes momentarily positive.After threshold is reached, the sodium ions move into the cell via the Na+/K+ pump, the inside of the cell becomes momentarily positive. At almost the same time as Na+ channels are closing, voltage-gated potassium channels open, allowing an increased movement of potassium ions out of the cellAt almost the same time as Na+ channels are closing, voltage-gated potassium channels open, allowing an increased movement of potassium ions out of the cell The…