Describe the molecular mechanisms of hippocampal LTD at the introduction level (Introduction to Neuroscience College Level). Provide diagrams from the web or from a textbook or a website to complement your explana
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Describe the molecular mechanisms of hippocampal LTD at the introduction level (Introduction to
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- Introduction to Neuroscience Which of the following does NOT contribute to the pathology of Alzheimer’s disease? Group of answer choices beta amyloid plaques neurofibrillary tangles excess activity in the hippocampus loss of acetylcholine containing neurons all of the above contribute Please explain your explain full detail and explain your reasoning in detail.Describe hippocampal place cells AT THE COLLEGE LEVEL OF THE INTRODUCTION TO NEUROSCIENCE COURSE Provide diagrams and other visualizations to explain this question.Parkinson's disease affects the cerebral basal ganglia, resulting in tremors in limbs, slowness in beginning and completing movements, and other abnormalities of muscle control. Explain what the root cause of Parkinson's disease is (where, what is happening and what symptoms are seen). A treatment option is Dopamine, that usually fails. Propose a reason why this treatment fails. Propose a biomedical device intervention that you could use to control symptoms and how it works.
- Describe the symptoms, pathology, and treatment of Alzheimer’s Disease at the introduction level (Introduction to Neuroscience College Level). Provide diagrams to complement your explanation.Describe neurophysiological control of skill by tracing likely sensory, motor, and CNS processing pathways. Don't copy from GoogleBrain function is monitored by an electroencephalogram (EEG) True or false?
- Histological and brain imaging methods can be used to detect or confirm Alzheimer’s disease. Select ALL that apply: Functional MRI imaging of neuronal activity during episodic memory tasks shows increased activity in the hippocampus. The sulci appear larger The gyri appear thicker The lateral ventricles are enlarged Functional imaging shows a decreased metabolic activity in the temporoparietal cortex at resting states indicating defects in episodic memoriesPhenytoin (sodium channel blocker) and ethosuximide (calcium channel blocker) are anti-seizure drugs that stop seizures from happening. These drugs work by inhibiting electrical impulses (action potentials) from occurring. Explain the importance of sodium and calcium channels on a neuron and and the reasons why action potentials do not occur when these channels are inhibited. Be sure to include the phases of an action potential, the channels involved, and the importance of an action potential. Be detailed in your explanationPhenytoin (sodium channel blocker) and ethosuximide (calcium channel blocker) are anti-seizure drugs that stop seizures from happening. These drugs work by inhibiting electrical impulses (action potentials) from occurring. Explain the importance of sodium and calcium channels on a neuron and and the reasons why action potentials do not occur when these channels are inhibited. Be sure to include the phases of an action potential, the channels involved, and the importance of an action potential.
- (Max of 10 sentences only. Paragraph style only. No bullet points. Feel Free to include visuals as well.) Provide descriptions on the structural and functional characteristics of the nervous system pathways that convey sensory information versus motor information.Explain how the signal transmission at a synapse in an individual with Parkinson's disease is different than an unaffected individual. List the steps involved in an action potential moving from the axon terminal of the pre-synaptic neuron to the dendrites of the post-synaptic neuron. Explain how the process is different in individuals affected with Parkinson's disease.Intro to Neuroscience: Which of the following is an effect of cocaine and amphetamine? Group of answer choices Blocking dopamine reuptake Activating acetylcholine receptors on VTA neurons Directly activating dopamine receptors Inhibiting GABA release from interneurons All of the above