Give an account of the excitation-contraction coupling (Figure 3) in skeletal muscle fibers.Highlight the role of dihydropyridine receptors and ryanodine receptors and the fate ofA detailed description of the cross bridge cycle is notcalcium ions after contraction.necessary.-Axon teminal of(=)somatic motor neuronAChMuscle fiberpotentialPion++Action potential-NaMotor end plate-T-tubuleSarcoptasmic reticulumlellllCa2+DHPreceptorTropomyosinTroponinz diskActinM lineMyosinheadMyosin thick filament

Each skeletal muscle fiber is a single cylindrical muscle cell. An individual skeletal muscle may be…

Give an account of the excitation-contraction coupling (Figure 3) in skeletal Highlight the role of dihydropyridine receptors and ryanodine receptors and the fate of calcium ions after contraction. A detailed description of the cross bridge cycle is not necessary. -Axon temminal of somatic motor neuron (m) ACh Muscle fiber பப்பு Action potential- Na Motor end plate- T-tubule Sarcoplasmic roticulum lelll DHP receptor Tropomyosin Troponin Z disk Actin M line Myosin head Myosin thick filament

Give an account of the excitation-contraction coupling (Figure 3) in skeletal Highlight the role of dihydropyridine receptors and ryanodine receptors and the fate of calcium ions after contraction. A detailed description of the cross bridge cycle is not necessary. -Axon temminal of somatic motor neuron (m) ACh Muscle fiber பப்பு Action potential- Na Motor end plate- T-tubule Sarcoplasmic roticulum lelll DHP receptor Tropomyosin Troponin Z disk Actin M line Myosin head Myosin thick filament

Human Physiology: From Cells to Systems (MindTap Course List)

9th Edition

ISBN:9781285866932

Author:Lauralee Sherwood

Publisher:Lauralee Sherwood

Chapter8: Muscle Physiology

Section: Chapter Questions

Problem 4TAHL

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Similar questions

In the diagram below, label the fine structure of a muscle, down to one of its myofibrils. Identify the basic unit of contraction in a myofibril.
The muscle relaxes if no new nerve signal arrives. However the neurotransmitter from the previous stimulation is still present in the synapse The activity of helps to remove this neurotransmitter. myosin action potential tropomyosin acetylcholinesterase
Every skeletal muscle fiber is supplied by a motor neuron at the NMJ. Watch this video (http://openstaxcollege.org/l/skelmuscfiber) to learn more about what happens at the neuromuscular junction. (a) What is the definition of a motor unit? (b) What is the structural and functional difference between a large motor unit and a small motor unit? Can you give an example of each? (c) Why is the neurotransmitter acetylcholine degraded after binding to its receptor?
Watch this video (http://openstaxcollege.org/l/micromacro) to learn more about macro- and microstructures of skeletal muscles. (a) What are the names of the junction points between sarcomeres? (b) What are the names of the subunits within the myofibrils that run the length of skeletal muscle fibers? (c) What is the â€œdouble strand of pearlsâ€� described in the video? (d) What gives a skeletal muscle fiber its striated appearance?
Explain why (a) calcium ions and (b) ACh are vital for muscle contraction.
The release of calcium ions initiates muscle contractions. Watch this video (http://openstaxcollege.org/l/calciumrole) to learn more about the role of calcium. (a) What are â€œT-tubulesâ€� and what is their role? (b) Please also describe how actin-binding sites are made available for cross-bridging with myosin heads during contraction.
What is the function of the sarcoplasmic reticulum in muscle cell contraction?
After death, a person no longer makes ATP, so calcium stored in the sarcoplasmic reticulum diffuses down its concentration gradient into the muscle cytoplasm. This result is rigor mortis----an unbreakable state of muscle contraction that stiffens the body for a few days until muscles begin to decay. Explain why this contraction occurs.
Nerve impulses first stimulate a skeletal muscle fiber at _______. a. T tubules b. sarcomeres c. neuromuscular junctions d. actin binding sites