High levels of caffeine are known to make people "jittery" or have shakey movements. Tests of people who have taken large doses of caffeine have shown increased amounts of Ca+ in their blood. Provide a hypothesis on what caffeine is doing to promote jittery movements?
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- Someone goes for a jog first thing in the morning and encounters a squadron of javelinas, causing a release of epinephrine. Some of this circulating epinephrine binds to a G-alpha-s coupled receptor (GPCR) on the surface of muscle cells. Which of the following would occur in the muscle cell as a result of this activation? Nitric oxide synthesis would increase. cyclic AMP (cAMP) activity would increase. Phospholipase C (PLC) would hydrolyze (cut) the molecule PIP2. The levels of adenylate cyclase activity would decrease. The levels of adenylate cyclase activity would decrease. Which ligand is CORRECTLY matched with its characteristic? Group of answer choices Glucagon: stimulates the activation of the mitogen activated protein (MAP) kinase pathway Nitric oxide (NO): must bind to its target protien withn a few seconds because NO degrades rapidly Steroid hormone: produces a response in its target cells within seconds Acetylcholine: hydrophobic molecule that binds to…Sarin is an inhibitor of acetylcholinesterase. Draw a mechanism that shows this.When the neurotransmitter acetylcholine (ACh) binds the acetylcholine receptor (a GPCR) on muscle cells, it causes them to contract. ZIGGY, a chemical analog of ACh, also binds to the same acetylcholine receptor on muscle cells, but instead causes the muscle cells to relax. For this reason, it is sometimes prescribed as a muscle relaxer. Explain in 3-4 sentences how ZIGGY could cause muscle relaxation. How can both ZIGGY and ACh bind the same GPCR? And then how can they have different effects on the cells, despite binding to the same receptor on the same cells?
- Construct a mechanism where sarin inhibits acetylcholinerase.Botulinum toxin (Botox) works by blocking the exocytosis of acetylcholine. A number of different medical and cosmetic procedures use this chemical. Injection of small quantities of botulinum toxin (Botox) into specific overactive muscles causes muscle relaxation that smooths the skin and reduces wrinkles. Injection into certain spinal ganglia helps reduce a certain form of chronic pain, and injections into the salivary glands causes the reduction of overactive saliva production. What type of toxin is Botox? a. Agonist b. AntagonistBotulinum toxin (Botox) works by blocking the exocytosis of acetylcholine. A number of different medical and cosmetic procedures use this chemical. Injection of small quantities of botulinum toxin (Botox) into specific overactive muscles causes muscle relaxation that smooths the skin and reduces wrinkles. Injection into certain spinal ganglia helps reduce a certain form of chronic pain, and injections into the salivary glands causes the reduction of overactive saliva production. What type of toxin is Botox? Agonist or Antagonist
- Heroin (diamorphine) is: O an opioid receptor antagonist. O a long-acting opioid agonist that produces dysphoria. O a drug that is metabolised in humans to morphine. a drug with high liability for physical dependence and low liability for psychological dependence. Question 4 Cocaine exerts sympathomimetic activity by: O acting directly as an agonist at alpha 1-adrenoceptors. inhibiting the re-uptake of noradrenaline at axon terminal sympathetic neurons. O inhibiting alpha 1-adrenoceptors. O inhibiting the breakdown of noradrenaline by monoamine oxidase.Serotonin, norepinephrine, and dopamine are a group of neurotransmitters collectively called monoamines. A lack of these monoamines has been linked to depression. Depression is treated with dru that prolong the effect of these neurotransmitters either by preventing the re-uptake of the neurotransmitters, or by inhibiting the enzyme that breaks down these neurotransmitters. Which of the following statements about neurotransmitters'are true? The table lists the classifications of three antidepressants. Antidepressant Classification fluoxetine selective serotonin re-uptake inhibitor (SSRI) phenelzine monoamine oxidase (MAO) inhibitor amitriptyline tricyclic antidepressant Check all that apply. View Available Hint(s) MAO inhibitors are examples of monoamine antagonists. EFluoxetine inhibits the enzyme that breaks down monoamines. ODopamine plays a role in feelings of pleasure and pain. DA deficiency of serotonin norepinephrine, and dopamine is associated with depression. Some drug…Parkinson's Disease Parkinson's disease is a neurological degenerative disorder that affects movement. Most people affected with Parkinson's disease demonstrate rigidity, slow movement, and shaking. The symptoms of Parkinson's disease occur when the cells that produce dopamine neurotransmitters die in the brain. Since most symptoms of Parkinson's disease are caused by insufficient dopamine in the brain, many Parkinson's drugs either temporarily replenish dopamine or mimic the action of dopamine. Explain how the signal transmission at a synapse in an individual with Parkinson's disease is different than an unaffected individual. 1. List the steps involved in an action potential moving from the axon terminal of the pre-synaptic neuron to the dendrites of the post-synaptic neuron. 2. Explain how the process is different in individuals affected with Parkinson's disease.
- Thermoreceptors in the hypothalamus sense the temperature of the blood. When blood temperature begins to decrease, the hypothalamus releases TRH, which stimulates the release of TSH from the anterior pituitary gland. TSH stimulates the thyroid gland to release thyroid hormone (TH). TH stimulates an increase in body temperature, which causes the hypothalamus to decrease the release of TRH. In this reflex pathway, what is the response? A) Increased body temperature B) Increased release of TH C) Thermoreceptors D) Decreased release of TRHA patient comes into a hospital emergency room sedated and non-responsive. She had been at a college party where she drank many shots of vodka. What is the most likely explanation for her behavioral and physiological symptoms? A. The alcohol increased the actions of GABA at the GABA receptor chloride channel B. The alochol increased dopamine and serotonin activity by running the transporters backwards C. The alcohol blocked the dopamine reuptake transporter D. the alcohol activated serotinin autoreceptors in the brain E. none of the aboveDuring a sympathetic nervous system response to an event, your heart rate increases, indicating the part of the heart responsible for controlling heart rate (the sinoatrial node) is innervated by sympathetic nerves. The SA node is also responsive to epinephrine,a hormone secreted by the adrenal medulla. Explain why the onset of the effect of the hormone is so much slower than the onset of the effect from sympathetic nerve stimulation and yet the effect of the hormone lasts much longer than the effect of nerve stimulation.