In a dramatic debate that helped to define neuroscience during the Victorian era, two researchers held opposing views about neuronal communication. Although it took time to determine an answer, who was credited with propos the correct explanation of synaptic transmission? O Santiago Ramón y Cajal O Charles Bell O Camillo Golgi O François Magendie

In a dramatic debate that helped to define neuroscience during the Victorian era, two researchers held opposing views about neuronal communication. Although it took time to determine an answer, who was credited with propos the correct explanation of synaptic transmission? O Santiago Ramón y Cajal O Charles Bell O Camillo Golgi O François Magendie

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Biology (MindTap Course List)

11th Edition

ISBN:9781337392938

Author:Eldra Solomon, Charles Martin, Diana W. Martin, Linda R. Berg

Publisher:Eldra Solomon, Charles Martin, Diana W. Martin, Linda R. Berg

Chapter41: Neural Signaling

Section: Chapter Questions

Problem 6TYU: A presynaptic neuron in the cerebrum transmits information to hundreds of other neurons. This...

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I have stimulated a neuron with dopamine for a second followed by treating it with cocaine and calcium chelating agent (which chelates out all calcium from the system making them unavailable for function), what will be the effect of such impulse on generating an action potential in the post-synaptic neuron? Will it be different if I pretreat the set of nerve cells with cocaine and calcium chelating agent followed by stimulating the neuron with dopamine? Justify your answer with proper reasoning in brief.
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Researchers have designed a neuron that's unable to produce and release neurotransmitters. Can that neuron still have an action potential? Choose the answer with the correct answer and the correct reasoning. O No, action potentials need neurotransmitters to increase message speed O No, a neuron can't receive messages without its own neurotransmitters O No, a lack of neurotransmitters would leave a neuron unmyelinated O No, action potentials don't occur in the axon O Yes, action potentials don't require neurotransmitters:
Which of the following statements is true about inhibitory synapses (IPSPs)? inhibitory neurotransmitters make the post-synaptic neurons more likely to have an action potential inhibitory neurotransmitters make the receiving cell less likely to have an action potential inhibitory synapses are calming inhibitory neurotransmitters are never good as they always inhibit important life processes
The limbic system utilizes specific pathways in the brain to prevent sensory overload and help with alertness. O False, the limbic system is involved in emotions and maps to one location in the diencephalon O False, the limbic system is involved in human emotion and is comprised of pathways primarily located in the reticular activating system (RAS). O True O None of these options are correct
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In 2003, the Nobel Prize in Physiology or Medicine wasawarded to Paul C. Lauterbur and Sir Peter Mansfield fordiscoveries related to magnetic resonance imaging (MRI).This is a tool to see the structures of the body (not just thenervous system) that depends on magnetic fields associatedwith certain atomic nuclei. The utility of this technique inthe nervous system is that fat tissue and water appear asdifferent shades between black and white. Because whitematter is fatty (from myelin) and gray matter is not, theycan be easily distinguished in MRI images. Visit the NobelPrize website (http://openstaxcollege.org/l/nobel_2) toplay an interactive game that demonstrates the use of thistechnology and compares it with other types of imagingtechnologies. Also, the results from an MRI session arecompared with images obtained from x-ray or computedtomography. How do the imaging techniques shown in thisgame indicate the separation of white and gray mattercompared with the freshly dissected tissue…
71) Figure below illustrates the data from one of the original experiments that showed evidence for 'silent synapses'. Stimulation of CA3 axons resulted in no postsynaptic response when the CA1 neuron was voltage-clamped at -60 mV. +30 mV 30 response amplitude (PA) 20 10 Isnos 0 -10 -20 -60 mV 0 100 200 300 stimulus number AP5 400 600 700 a) Why was the postsynaptic CA1 neuron voltage clamped to +30 mV? (a) To stimulate insertion of AMPA receptors into the postsynaptic membrane (b) To move NMDA receptors into the synapse from an extrasynaptic site (c) To cause coordination between the pre- and postsynaptic neuron (d) To increase conductance of the NMDAR by removing the Mg2+ block (e) To stimulate AMPA receptors, which in turn activate NMDA Rs b) Explain the results obtained when AP5 was applied while the neuron was held at +30 mV. What is the interpretation of the result? (2-3 sentences.
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