Which of the following is involved in the formation of cancer? O mutations in genes involved in DNA repair processes active telomerase that prevents the shortening of telomeres mutations in proto-oncogenes and/or tumor suppressor genes O all the above

Which of the following is involved in the formation of cancer? O mutations in genes involved in DNA repair processes active telomerase that prevents the shortening of telomeres mutations in proto-oncogenes and/or tumor suppressor genes O all the above

Name: 6 Which of the following is involved in the formation ofcancer?O mutat
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Description: 6 Which of the following is involved in the formation ofcancer?O mutations in genes involved in DNA repair processesactive telomerase that prevents the shortening of telomeresmutations in proto-oncogenes and/or tumor suppressor genesO all the above

Biology (MindTap Course List)

11th Edition

ISBN:9781337392938

Author:Eldra Solomon, Charles Martin, Diana W. Martin, Linda R. Berg

Publisher:Eldra Solomon, Charles Martin, Diana W. Martin, Linda R. Berg

Chapter17: Developmental Genetics

Section: Chapter Questions

Problem 8TYU: Which of the following statements about cancer is false? (a) oncogenes arise from mutations in...

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Benign tumors: a. are noncancerous growths that do not spread to other tissues b. do not contain mutations c. are malignant and clonal in origin d. metastasize to other tissues e. none of these
are changes to the order of nucleotides in a segment of DNA that codes for a protein. Proto-oncogenes Tumor suppressor genes Gene mutations Negative regulators
Which of the following statements about cancer is false? (a) oncogenes arise from mutations in proto-oncogenes (b) tumor suppressor genes normally interact with growth-inhibiting factors to block cell division (c) more than 120 cancer-driving genes have been discovered (d) oncogenes were first discovered in mouse models for cancer (e) the development of cancer is usually a multistep process involving both oncogenes and mutated tumor suppressor genes
Tumor suppressor genes _____. a. occur normally in cells b. promote metastasis c. are brought into cells by viruses d. only rarely affect the development of cancer
Which of these events do you think is likely to promote tumorigenesis in affected cells? Group of answer choices DNA methylation of the promoter region of a tumor suppressor gene Hypermethylation of transcription factors that antagonize Wntsignaling Open chromatin in the region of of DNA harboring an activating mutation of beta-catenin All of these
Which one of the following events is unlikely to beassociated with cancer?a. mutation of a cellular proto-oncogene in a normaldiploid cellb. a chromosomal translocation with a breakpointnear a cellular proto-oncogenec. deletion of a cellular proto-oncogened. mitotic nondisjunction in a cell carrying a deletionof a tumor-suppressor genee. incorporation of a cellular oncogene into a retroviruschromosome
Of the following choices a loss of function mutation would most lukely contribute to cancer onset if the mutant gene codes for a : telomerase tumor suppressor protein ATP synthesis enzyme RNS binding protein
Which of the following do cancer cells rely on to be able to divide indefinitely? tumor suppressors proto-oncogenes telomeres telomerase
The product of the human papillomavirous oncogene E7promotes cancer by emhancing expression of telomere inhibiting DNA damage repair pathways binding to Rb protein and preventing itd function ubiquitinating the tumor suppressor p53
All of the following are characteristics of oncogenes EXCEPT Multiple Choice they are mutated proto-oncogenes. they cause cancer. they are genes that stimulate uncontrolled cell division. they are tumor suppressor genes.
The Bax gene, codes for a cytosolic protein that plays an important role in apoptosis. Growth factor withdrawal stimulates the pro-apoptotic protein Bax. Which of the following are true for the Bax gene? Select all that apply The Bax gene is a proto-oncogene The Bax gene is a tumor suppressor gene In cancer or transformed cells, the Bax gene mutation would be dominant, and a gain of function mutation In cancer or transformed cells, the Bax gene mutation would be recessive, and a loss of function mutation
Products of proto-oncogenes and tumor-suppressor genes can be growth factors and growth factor receptors signaling molecules like protein kinases proteins that control DNA repair transcription factors all of the above