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A: Omega-3 fatty acids: it is an essential fatty acid that must be taken in diet.
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A: Introduction:- There are three steps of cell signalling. 1. Reception: A signalling molecule from o...
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A: Introduction: Cell division is the means of reproduction in which all cells need to copy their chrom...
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A: Introduction :- A homotropic allosteric modulator is both a substrate and a regulatory molecule for ...
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A: Features associated with Homo habilis except-
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A: Replication is the process of producing two same copies of entire genome.
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A: Cancer is a disease in which cells of the body multiply uncontrollably and form tumours. Cancer is ...
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Q: Explain and list down the protocols in doing or preparation of materials for polymerase chain reacti...
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Q: What is tej role of indigenous knowledge in the development of indigenous science?
A: Answer
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Q: One of the features of the Acheulian stone tool industry is that the tools were made from stone that...
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A: The activity of allosteric proteins/enzymes is explained by two prominent models. Monod, Wyman, and ...
Q: Zhoukoudian Cave provides clear evidence that Group of answer choices A: Homo erectus ventured as fa...
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Q: 45. Refer to the drawings in Figure 13.2 of a single pair of homologous chromosomes as they might ap...
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Question:-
5. What happens when p53 senses that DNA damage is beyond repair? Why is this
advantageous?
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- Part || – An Unexpected Visitor After Ivy was finished meeting with Dr. Alvarez, she didn't feel like going back to her dorm room where her room- mates were waiting to hear from her. She decided to process her diagnosis at the local coffee shop. Curling up in an oversized chair with a warm cookie and peppermint hot cocoa with whipped cream always made her feel better. Sitting alone would give her time to think. How could something so important go so wrong in her cells? How did cells get or make the mitochondria anyway? Obviously, cells could survive without mitochondria if they had to, so why had mitochondria evolved in the first place? Just then, Ali, a fellow student in her biology class who made it a point to sit near her each class, walked over to strike up a conversation. "Hi Ivy! Whať's up?" Trying to avoid conversation, Ivy simply shrugged and mumbled, “Not much." Ali gave her a big smile and said "I've noticed that you take good notes in bio, would you want to study together...…MM, a 54-year old female presents to the Family Medicine Clinic due to a 2-week history of increasing shortness of breath and cough and mild/moderate pain in left side. She was diagnosed with infiltrating intraductal adenocarcinoma of the left breast 5 years ago; at that time, ER(-)/ PR(-); her-2/neu(+); p53(+); staged as having T3N1M0, stage IIIA, high-risk breast cancer. She underwent a modified radical mastectomy with axillary node dissection followed by 6 cycles of CMF chemotherapy. Her mother and sister also had a history of breast cancer. Past Medical History Gravida 4, para 4; menses onset age 13; HTN x 10 years; Type 2 DM x 8 years; breast CA described above; remained disease free until present follow up. Past Surgical History: Left modified radical mastectomy 5 years ago; cholecystectomy 14 years ago. Medications: Glyburide, 5mg PO BID Verapamil SR, 240mg PO daily Furosemide, 40mg PO daily Allergies: NKDA Physical Examination: GEN: Well-developed, obese woman in no…4:17 Regarding chronic disease prevention, match each term with the phrase that best describes it Molecule produced by innate immune cells. that is involved in maintaining chronic inflammation A protein produced by the liver that is al frequently used marker of chronic inflammation The degree to which a certain food elevates blood glucose after it's eaten A small molecule that can bind to histones and DNA, frequently with the result of gene silencing An enzyme that rebuilds the end caps of chromosomes after each cell division, thus prolonging the life of the cell [Choose] [Choose] Interleukin-6 TNF-alpha Methyl Tag Glycemic Load C Reactive Protein Glycemic Index Telomerase [Choose] [Choose ] [Choose] X
- Which molecule is a Cdk inhibitor that is controlled by p53? cyclin anti-kinase Rb p21Drug 2-Lumacaftor (VX-809): In people with the most common CF mutation, F508del, a series of problems prevents the CFTR protein from folding into the correct shape and reaching its proper place on the cell surface. The cell recognizes the protein as abnormal and targets it for degradation before it makes it to the cell surface. In order to treat this problem, two drugs are required - an agent to get the protein to the surface and then Ivacaftor to open the channel and increase chloride transport. The drug Lumacaftor has been identified as a treatment to help with the trafficking of the protein to the cell surface. When Lumacaftor is added to Ivacaftor, the protein gets to the surface and also increases chloride transport by increasing channel opening time. For which class(es) of mutations would Lumacaftor be most effective?Explain the factors that reactivate the dormant herpes simplex virus (2 pts) How can a defect in p53 gene contribute to cancer development? (2 pts)
- p53 is a gene / protein often associated with cancer. Why? What does p53 do? What kind of gene is it? Is it associated more with any one particular type of cancer or all cancers? Tell me more about p53, but please do not exceed one typed page./20. In class, we discussed diffferent types of genetic change that can cause a normal gene (proto- oncogene) to become a cancer-causing gene (oncogene), Which of the following would not be a cause? A) translocation or transposition (movement of DNA within the same genome) B) gene ampliffication (increased number of copies of a given gene) C) epigenetic change D) point mutation that changes the gene's product E) loss of telomeres during DNA replication ancer?Q6\ How can cell prevent any error can cause cancer or abnormal cell?
- What is the rationale for synthesizing and rapidly degrading p53 protein in the cell? please give me new answerHi, can you elaborate about the following: certain characterize mutations in p63 and compare that to known mutations in p53.Compare and contrast the intrinsic and extrinsic apoptosis pathways. Please keep brief - 3 sentences/dot points each.
