Chronic periodontitis has been identified as the most prevalent form of periodontitis affecting the adults and occasionally the adolescents and children.(Merin, 2015) This periodontal disease of inflammatory origin is thought to be the result of host immune response against bacterial colonies in the subgingival plaque.(Merin, 2015) In many cases, systemic and environmental factors play a significant role in the disease progression. Common risk factors such as uncontrolled diabetes mellitus and smoking, contribute to periodontal destruction in genetically susceptible individuals by modifying the host’s immune reactions towards periodontal pathogens. Clinically, the diagnosis of chronic periodontitis is made based on the presence of supragingival and subgingival plaque and calculus, signs of gingival inflammation, periodontal attachment loss and radiographic bone loss. Chronic periodontitis can be further classified based on the extent and severity of periodontal destruction. Localised chronic periodontitis is diagnosed when less than 30% of the sites are affected; whereas the generalised form of the disease affects more than 30% of the sites. The severity of the disease is determined by the amount of clinical attachment loss. It is classified as mild, moderate or severe when 1-2mm, 3-4mm or 5mm or more attachment loss is recorded respectively. (Merin, 2015) The treatment of chronic periodontitis involves a sequence of therapeutic procedures, which is termed periodontal
Periodontal disease is more commonly known as gum disease or gingivitis. This infection is serious enough, that it can lead to tooth loss if left untreated. This chronic infection starts around the tooth and it affects the supporting bone and gums. Periodontal disease can affect anywhere from one tooth to all thirty-two teeth. The disease pathology starts with the plaque that builds up on your teeth everyday.
Treatment of periodontal disease can be broadly divided into two, non-surgical and surgical. Longitudinal comparison between non-surgical and surgical therapy had been studied extensively by groups of researchers, namely the Gothenburg, the Michigan, the Minnesota, the Nebraska, and the Arizona group. There are some heterogeneities between studies from different groups. Some studies focused on single rooted teeth, while others included molars. Majority of the studies are done in a university setting, while the Arizona study was done in private practices.
Some studies have been conducted but the World Workshop on Periodontics stated that controlled clinical trials that evaluated the role that occlusion had on the progression of periodontal disease in humans, was unethical. To avoid unethical situations, patient records from a private practice facility were collected and studied to see if there was a connection between occlusal discrepancies and the progression of periodontal disease. The records that were studied were from patients that had periodontal evaluations as well as occlusal assessments. All of the patients studied had periodontal disease but only some of them had occlusal decencies. After a twelve month period some patients returned and had another periodontal evaluation and occlusal assessment and the data was compared to the data that was collected twelve months prior. The data collected was compared. Patients without occlusal discrepancies and patients with occlusal discrepancies both had worsening periodontal disease after twelve months of no treatment but, the progression of periodontal disease and increased pocket probing depths
If you suffer from advanced periodontal disease, when discussing treatment options with your dentist, make sure that you also talk about the different recovery times that go with each procedure. Make sure that you are able to set aside the proper amount of time for your mouth to heal based on the treatment option you
Gum diseases (sometimes called PERIODONTAL or GINGIVAL DISEASES) are infections that harm the gum and bone that hold teeth in place. When plaque stays on your teeth too long, it forms a hard, harmful covering, called TARTAR, that brushing doesn't clean. The longer the plaque and tartar stay on your teeth, the more damage they cause. This is called GINGIVITIS. If gingivitis is not treated, over time it can make your gums pull away from your teeth and form pockets that can get infected. This is called PERIODONTITIS. If not treated, this infection can ruin the bones, gums, and tissue that support your teeth. In time, it can cause loose teeth that your dentist may have to remove.
Research shows that periodontitis might be connected with the development of cardiovascular disease. One theory is that the inflammatory proteins and the microorganisms in the periodontal tissue enter the circulatory system and cause different consequences for the cardiovascular system. Researchers know that inflammation leads to hardened arteries, called atherosclerosis. That is a condition that
Chronic periodontitis is a slowly progressive inflammatory disease that results in the destruction of periodontium (Armitage and Cullinan, 2010, Shaddox and Walker, 2010). It is characterized by clinical attachment greater than or equal to 5mm and radiographic evidence of alveolar bone loss (Cabanilla, Neely and Hernandez, 2008). Teeth with active periodontal
Advanced periodontitis is the final stage of gum disease, where the pockets around your teeth deepen, and the bone and fibres supporting your teeth are mostly destroyed. This can cause teeth to move or loosen and can affect the way you bite. At this stage, of aggressive professional treatment is unable to save them, they may need to be
Cigarette smoking not only has a negative effect on respiratory and cardiovascular health, but also has been associated with diabetes, gastrointestinal and thyroid diseases, and almost every other organ system in the body (Johnson & Guthmiller, 2007). Linked to various cancers, smoking is extremely detrimental (Johnson & Guthmiller, 2007). Smoking is also “the major risk factor for periodontitis” (Preshaw et al, 2015). While increasing the prevalence of periodontitis, smoking also has a profound effect on the severity of periodontal disease in individuals (Preshaw et al, 2015). The third National Health and Nutrition Examination Survey (NHANES III) in 2000 found that “41.9% of periodontitis cases in the United States were attributable to current cigarette smoking” (Tomar & Asma, 2000). Smokers have a four times greater chance to be diagnosed with periodontitis (Johnson & Guthmiller, 2007). Indeed, patients who are smokers also do not appear to respond as favourably to periodontal treatment and show a greater risk of refractory and/or recurrent periodontal disease (Fardal, 2008). Multiple studies show that smoking has the potential to alter numerous processes in the oral cavity (Preshaw et al, 2015). Most notably, smoking has been shown to modify various aspects of physiology in patients, impair immunological response and alter the microflora of the oral cavity (Presahaw et al, 2015).
Periodontitis is an infectious disease that affects all organs of the tooth structure: gums, root surface, bone and ligaments that tooth is attached to the bone. Periodontitis begins inconspicuously as gingivitis, which is manifested mainly by bleeding. Later, the inflammation spreads to the depth and results in a progressive loss of bone. Gums begin to recede, the roots of the teeth is exposed arise periodontal swelling and pus. Teeth start to wobble, spacing and release. The decisive factor is the quality of oral hygiene, not
BACKGROUND: Aggressive periodontitis (AgP) by definition is a group of rare, very destructive forms of periodontitis, usually manifests itself at an early age and shows a marked tendency to aggregate in families. A variety of factors, such as microbial, environmental and behavioral have been suggested as risk factors of Aggressive periodontitis.
Gingivitis, the acute inflammatory phase of periodontal disease, is characterized by redness, bleeding and swelling of the gingival tissue, and if let untreated can progress into periodontitis characterized by the formation of gingival pockets and potential tooth loss. According to the Centers for Disease Control and Prevention (CDC), periodontitis affects nearly half (47%) of American adults, which translates to nearly 65 million patients over the age of
People who can help treat the disease are the dentist, periodontist, or the dental hygienist. One of the first steps for treatment is the deep cleaning which is normally called scaling and root planing. First they will usually start with scaling the teeth which removes all the tarter from under the gum line then they will start root planing which gets rid of the rough spots on the tooth root where germs gather (“Periodontal Disease”). Sometimes the dentist or periodontist will prescribe medications to help with the inflammation. Some might be used before the deep cleaning because the dentist or periodontist is scraping up bacteria. If the periodontal disease progresses into a worsen state surgery may be and options that should be
(178) published a paper in the form of a clinical trial in order to examine the effect of periodontal treatment on the biological and clinical parameters of RA. Their proposed study was randomized controlled trial including participants with both RA and periodontitis. The investigators plan to involve a total of 40 individuals into two groups (intervention group including full-mouth SRP, followed by systemic antibiotics, amoxicillin or clindamycin, if allergic to penicillin, for seven days, oral hygiene instructions, and rinsing with 0.12 % chlorhexidine gluconate for 10 days after periodontal treatment). Patients will be followed for three months, and the same intervention will then be applied to the control group. The primary outcome of this study was a change in DAS28 score by decreasing RA activity. A major drawback of this study is the use of amoxicillin or clindamycin adjunctive to SRP. This antimicrobial approach should not be used as periodontal pathogens have been shown to be resistant to these
The long-term treatment plan for managing chronic periodontitis should include periodic monitoring of periodontal status and appropriate maintenance procedures.28 Experimental studies have reported very successful treatment outcomes when patients are professionally maintained at two-week intervals,161 but such a program is impractical for most chronic periodontitis patients. Therefore, to maximize successful therapeutic outcomes, patients must maintain effective daily plaque control. It also appears that in-office periodontal maintenance at three to four month intervals can be effective in maintaining most